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HOME > Product search results > Code No. 311 Anti-GST-P pAb

Code No. 311

Anti-GST-P pAb

Price

¥32,000

Availability (in Japan)

10 or more

(In Japan at 17:00,
Nov 14, 2019 in JST)

Size

100 µL

Data
  • Immunohistochemistry

Clonality Polyclonal Clone Polyclonal
Isotype (Immunized Animal) Rabbit IgG
Applications
WB
1:1,000-1:2,000  
IH
1:1,000-1:2,000 (Can be used in cryosections & paraffin-embedded sections.)  
Immunogen (Antigen) purified rat liver glutathione S-transferase P
Reactivity [Gene ID]

Human[2950], Mouse[14870], Rat[24426]

Storage buffer PBS/50% glycerol, pH 7.2
Storage temp. -20°C Conjugate Unlabeled Manufacturer MBL
Alternative names GSTP1 glutathione S-transferase pi 1, PI, DFN7, GST3, GSTP, FAEES3
Background Placental glutathione S-transferase (GST-P), a member of glutathione S-transferase, is known for its specific expression during rat hepatocarcinogenesis and has been used as a reliable tumor marker for experimental rat hepatocarcinogenesis.
Related products 311-H Anti-GST-P pAb
312 Anti-GST-π pAb
Citations

Western Blotting

  1. Hokaiwado N et al. Glutathione S-transferase Pi mediates proliferation of androgen-independent prostate cancer cells. Carcinogenesis 29, 1134-8 (2008)(PMID:18413363)

Immunohistochemistry

  1. Endoh T et al. Inhibition by acetylsalicylic acid, a cyclo-oxygenase inhibitor, and p-bromophenacylbromide, a phospholipase A2 inhibitor, of both cirrhosis and enzyme-altered nodules caused by a choline-deficient, L-amino acid-defined diet in rats. Carcinogenesis 17, 467-475 (1996)(PMID:8631132)
  2. Denda A et al. Inhibition by piroxicam of oxidative DNA damage, liver cirrhosis and development of enzyme-altered nodules caused by a choline-deficient, L-amino acid-defined diet in rats. Carcinogenesis 18, 1921-30 (1997)(PMID:9364001)
  3. Kobayashi Y et al. Prevention by 1'-acetoxychavicol acetate of the induction but not growth of putative preneoplastic, glutathione S-transferase placental form-positive, focal lesions in the livers of rats fed a choline-deficient, L-amino acid-defined diet. Carcinogenesis 19, 1809-14 (1998)(PMID:9806163)
  4. Ledda-Columbano GM et al. Cell proliferation induced by 3,3',5-triiodo-L-thyronine is associated with a reduction in the number of preneoplastic hepatic lesions. Carcinogenesis 20, 2299-304 (1999)(PMID:10590223)
  5. Zeng ZZ et al. Genetic resistance to chemical carcinogen-induced preneoplastic hepatic lesions in DRH strain rats. Cancer Res. 60, 2876-81 (2000)(PMID:10850431)
  6. Denda A et al. Increased expression of cyclooxygenase-2 protein during rat hepatocarcinogenesis caused by a choline-deficient, L-amino acid-defined diet and chemopreventive efficacy of a specific inhibitor, nimesulide. Carcinogenesis 23, 245-56 (2002)(PMID:11872629)
  7. Oyama K et al. Reduction of hepatocarcinogenesis by ursodeoxycholic acid in rats. Carcinogenesis 23, 885-92 (2002)(PMID:12016164)
  8. Nakaji M et al. IFN-alpha prevents the growth of pre-neoplastic lesions and inhibits the development of hepatocellular carcinoma in the rat. Carcinogenesis 25, 389-97 (2004)(PMID:14633663)
  9. Suzuki S et al. Specific differences in gene expression profile revealed by cDNA microarray analysis of glutathione S-transferase placental form (GST-P) immunohistochemically positive rat liver foci and surrounding tissue. Carcinogenesis 25, 439-43 (2004)(PMID:14656948)
  10. Sukata T et al. alpha(2)-Macroglobulin: a novel cytochemical marker characterizing preneoplastic and neoplastic rat liver lesions negative for hitherto established cytochemical markers. Am J Pathol. 165,1479-88 (2004)(PMID:15509519)
  11. asumura M et al. Promotion of thyroid carcinogenesis by para-aminobenzoic acid in rats initiated with N-bis(2-hydroxypropyl)nitrosamine. Toxicol. Sci. 86, 61-7 (2005)(PMID:15843508)
  12. Ueno S et al. Roxithromycin inhibits constitutive activation of nuclear factor {kappa}B by diminishing oxidative stress in a rat model of hepatocellular carcinoma. Clin. Cancer Res. 11, 5645-50 (2005)(PMID:16061884)
  13. Suzuki R et al. An animal model for the rapid induction of tongue neoplasms in human c-Ha-ras proto-oncogene transgenic rats by 4-nitroquinoline 1-oxide: its potential use for preclinical chemoprevention studies. Carcinogenesis 27, 619-30 (2006)(PMID:16219633)
  14. Takahashi M et al. Cellular distributions of molecules with altered expression specific to the tumor promotion process from the early stage in a rat two-stage hepatocarcinogenesis model. Carcinogenesis 29, 2218-26 (2008)(PMID:18586688)
  15. Tablas MB et al. Vitamin D3 suppresses the early stages of chemically induced hepatocarcinogenesis in rats: a dose-response analysis. Nutrire 43, 12 (2018)
  16. Seki K et al. Oral administration of fructose exacerbates liver fibrosis and hepatocarcinogenesis via increased intestinal permeability in a rat steatohepatitis model. Oncotarget 9, 28638-28651 (2018)(PMID:29983886)
  17. Alzahrani FA et al. Potential Effect of Exosomes Derived from Cancer Stem Cells and MSCs on Progression of DEN-Induced HCC in Rats. Stem Cells Int. 2018, 8058979 (2018)(PMID:30224923)
  18. Elkeiy MM et al. Chitosan nanoparticles from Artemia salina inhibit progression of hepatocellular carcinoma in vitro and in vivo. Environ Sci Pollut Res Int. (2018) In press.(PMID:30293105)
  19. Nishimura N et al. Acyclic retinoid and angiotensin-II receptor blocker exert a combined protective effect against diethylnitrosamine-induced hepatocarcinogenesis in diabetic OLETF rats. BMC Cancer. 18, 1164 (2018) (PMID:30477453)
  20. Ito Y et al. Expression Characteristics of Genes Hypermethylated and Downregulated in Rat Liver Specific to Nongenotoxic Hepatocarcinogens. Toxicol Sci. 169, 122-136 (2019)(PMID:30690589)
  21. El-Magd MA et al. Melatonin maximizes the therapeutic potential of non-preconditioned MSCs in a DEN-induced rat model of HCC. Biomed Pharmacother. 114, 108732 (2019)(PMID:30925457)
Product category
Research area
Cancer
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  • The availability is based on the information in Japan at 17:00, Nov 14, 2019 in JST.
  • The special price is shown in red color.
  • Please note that products cannot be ordered from this website. To purchase the items listed in this website, please contact us or local distributers.
  • Abbreviations for applications:
    WB: Western Blotting, IH: Immunohistochemistry, IC: Immunocytochemistry, IP: Immunoprecipitation
    FCM: Flow Cytometry, NT: Neutralization, IF: Immunofluorescence, RIP: RNP Immunoprecipitation
    ChIP: Chromatin Immunoprecipitation, CoIP: Co-Immunoprecipitation
    DB: Dot Blotting, NB: Northern Blotting, RNA FISH: RNA Fluorescence in situ hybridization
  • For applications and reactivity:
    *: The use is reported in a research article (Not tested by MBL). Please check the data sheet for detailed information.
    **: The use is reported from the licenser (Under evaluation or not tested by MBL).
  • For storage temparature: RT: room temparature
  • Please note that products in this website might be changed or discontinued without notification in advance for quality improvement.